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A growing body of evidence [8] suggests short sleep and OSA are associated with cardiometabolic disease risk factors including adiposity [9], hypertension [10,11], hypercholesterolemia [12,13], and hyperglycemia [14,15].
JMIR Form Res 2024;8:e54792
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Prolonged elevation of cortisol, a glucocorticoid, leads to increased adiposity, particularly abdominal adiposity, as well as increased appetite and preferences for high-fat and high-sugar foods [9]. Few studies have examined the association between cortisol and obesity among young children and most of this research has been cross-sectional [10-12].
JMIR Res Protoc 2024;13:e48549
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However, few studies have examined sex-based variations in adiposity and sleep duration using rigorous measures of adiposity [25,28-31]. Third, the association between adiposity and sleep on cardiometabolic outcomes requires further investigation, particularly considering updated blood pressure guidelines [32]. Fourth, adiposity, sleep, and cardiometabolic outcomes are influenced by a myriad of upstream and downstream factors [33].
JMIR Res Protoc 2021;10(3):e27139
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