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The subreddit data set yielded high androgens, high luteinizing hormone (LH), high anti-Mullerian hormone (AMH), and clinical evidence of metabolic issues. Thus, the subreddit users align with the PCOS laboratory result trends described in the literature. Tables 4 and 5 show that alignment was not perfect. However, this is expected, as even any 2 studies with slightly different inclusion and exclusion criteria are unlikely to exclusively achieve statistical equivalence across a range of factors.
JMIR Form Res 2023;7:e44810
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Abnormal secretion of gonadotropins, particularly LH, from the pituitary gland leads to abnormal and excessive ovarian theca cell androgens [59].
Estrogen and progestin components of COCs act together to suppress FSH and LH secretion and the midcycle gonadotropin surge by a feedback mechanism, which results in a decrease in ovarian steroidogenesis [49,60,61]. Indeed, suppression of LH is the major mechanism that mediates the effects of these products in PCOS patients [62].
JMIR Res Protoc 2018;7(4):e113
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